CVP and GI Pathology Exam 2 Practice

After a myocardial infarction, the damaged, noncontractile segments of the ventricle create a zone of endocardial injury with blood stasis, which promotes thrombus formation on the ventricular wall. A mural thrombus typically develops on the left ventricle after a transmural infarct, especially in a sizable anterior MI, during the subacute to early healing phase. This thrombus is often attached to the endocardium and may be pedunculated or broad-based; it is best seen with echocardiography. Embolization from a mural thrombus is a key complication, potentially causing systemic emboli such as stroke or peripheral vessel occlusion, which makes it a clinically important finding after MI.

Postural hypotension, while common in various contexts, is not a typical direct post-MI complication. An atrial septal defect is a congenital condition and not a consequence of myocardial infarction. Pulmonary embolism can occur in the broader setting of venous thromboembolism but is not the classic, typical finding specifically reflected as a complication of MI itself. The mural thrombus represents the classic post-MI change due to endocardial injury and stasis.